منابع مشابه
Satiety mechanisms in genetic risk of obesity.
IMPORTANCE A better understanding of the cause of obesity is a clinical priority. Obesity is highly heritable, and specific genes are being identified. Discovering the mechanisms through which obesity-related genes influence weight would help pinpoint novel targets for intervention. One potential mechanism is satiety responsiveness. Lack of satiety characterizes many monogenic obesity disorders...
متن کاملA survey of diet habit correction and physical activity in the treatment of overweight and obesity in children
Introduction: Obesity in children as a chronic problem with an increasing prevalence is one of the health problems in the last century. If the path from childhood to adolescent be accompanied by a gradual decline in physical activity and increased consumption of high-calorie foods, it will lead to obesity and a variety of chronic diseases. This study can help plan to reduce the prevalence and c...
متن کاملControversies in Obesity Treatment
The markedly high prevalence of obesity contributes to the increased incidence of chronic diseases, such as diabetes, hypertension, sleep apnea, and heart disease. Because of high prevalence of obesity in almost all countries, it has been the focus of many researches throughout the world during the recent decades. Along with increasing researches, new concepts and controversies have been emerge...
متن کاملObesity, Inflammation and Diet
Obesity is a state in which there is an over-accumulation of subcutaneous and/or abdominal adipose tissue. This adipose tissue is no longer considered inert and mainly devoted to storing energy; it is emerging as an active tissue in the regulation of physiological and pathological processes, including immunity and inflammation. Adipose tissue produces and releases a variety of adipokines (lepti...
متن کاملذخیره در منابع من
با ذخیره ی این منبع در منابع من، دسترسی به آن را برای استفاده های بعدی آسان تر کنید
ژورنال
عنوان ژورنال: British Journal of Nutrition
سال: 2002
ISSN: 0007-1145,1475-2662
DOI: 10.1079/bjn2002604